Along with the expanding list of potential epilepsy genes comes the challenge of genetic variants of uncertain significance (VUS).

VUS numbers are rising as more patients undergo NGS, but we lack the tools to determine VUS pathogenicity and disease mechanisms. To address these shortfalls, the EpiMVP will optimize cutting-edge multiplatform assays for epilepsy gene function.

A complex research diagram

The long-term goal of this project is to deliver an in vitro testing pipeline with defined phenotypes in human neuronal models to assay clinically relevant VUS for non-ion-channel epilepsy genes. Project 2 will test variants for the chosen genes in complex structural and functional assays, using human pluripotent stem cell (hPSC) knockout lines generated by the Human Epilepsy Tools Core (HETC). Human in vitro models will include 1) 2-D hPSC cultures: small molecule differentiation into excitatory or inhibitory cortical neurons, excitatory and inhibitory induced neurons (iNeurons) generated by forced transcription factor expression; and 2) Excitatory, inhibitory and fusion (combined excitatory and inhibitory) brain organoid cultures.

A complex research diagram

A series of microscopic images

Assays of VUS rescue (or deleterious gain-of-function) effects will include morphology, gene expression and neuronal/network activity. The latter includes multielectrode array [MEA] and patch clamp recordings, calcium imaging and depth electrode local field potential [LFP] recordings (in brain organoids).

Microscopic images and research results graphs

Our immediate goals are to optimize assays for 1-2 genes per year, determine VUS pathogenicity in vitro for these genes and, in concert with the Variant and Gene Curation Core (VGCC), refine the VUS list for further in vivo testing in Project 3. Two sets of milestones are proposed:

To determine VUS pathogenicity using 2-D hPSC assays (Milestone 1) and brain organoid models (Milestone 2). The HETC, Parent and Ross labs have experience generating cortical neurons via small molecule and iNeuron differentiation, and with multiple brain organoid culture protocols. We will express non-ion channel epilepsy gene variants chosen by the GVCC and Project 1, and via constructs generated by the HETC, in a knockout hPSC background. Assays will  include neuronal morphology, gene expression, calcium imaging and electrophysiology in 2-D and brain organoid cultures.

These studies will provide the following deliverables: 1) multiple optimized, cross-validated (between Parent and Ross labs) hPSC platforms to interrogate epilepsy genes; 2) determination of in vitro human neuronal VUS pathogenicity for at least 5 non-ion channel epilepsy genes; 3) human neuronal models for each epilepsy gene; and 4) optimized platforms for future mechanistic and precision therapeutic studies.

Participants

Jack Parent, MD

William J Herdman Professor, Research Professor

Department of Neurology, Michigan Neuroscience Institute

University of Michigan

M. Elizabeth Ross, MD, PhD

Professor, Feil Family Brain and Mind Research Institute

Department of Neuroscience, Department of Neurology

Weill Cornell Medical College

Lori Isom, PhD

Chair, Department of Pharmacology, Maurice H. Seevers Collegiate Professor of Pharmacology, Professor of Molecular and Integrative Physiology, Professor of Neurology

Department of Pharmacology

University of Michigan

Luis Lopez-Santiago, PhD

Associate Research Scientist

Department of Pharmacology

University of Michigan

Andrew Tidball, PhD

Research Investigator

Parent Lab

University of Michigan

Rajiv Dixit, PhD

Research Associate

Department of Neuroscience

Weill Cornell Medical College

Yukun Yuan, PhD

Research Assistant Professor

Department of Pharmacology

University of Michigan

Caroline Pearson, PhD

Research Associate

Department of Neuroscience

Weill Cornell Medical College

Tuo Ji, PhD

Research Specialist

Parent Lab

University of Michigan

Kyle Stokes, PhD

Postdoctoral Fellow

Department of Neurology and Michigan Neuroscience Institute

University of Michigan